Colin Wright continues his excellent string of explainers, this time on the Univariate Fallacy:
He goes on to talk about several different pieces deploying this fallacy — which he later describes as
One of his examples is a
New York Times editorial claiming that
"The Myth of Testosterone" is what fuels Caster Semenya's run (and the athletic superiority of men over women more generally):
Testosterone’s “authorized” biography, with its pat story about how it fuels male-typical athletic performance, is a powerful distraction from the hormone itself, occluding its fascinating, diverse and contingent actions within the body. Testosterone doesn’t drive a single path to athletic performance, nor even a small set of processes that can be linearly traced from more testosterone to more ability.
The idea that testosterone is the miracle molecule of athleticism, and, accordingly, that people with higher levels would obviously perform better, combines several beliefs: that “athleticism” is a kind of master trait that describes similar characteristics in different athletes, that “athletic performance” across different sports generally requires the same core skills or capacities, and that testosterone has a potent effect on all of them.
But that’s simply not true. The problem with trying to flatten athleticism into a single dimension is illustrated especially well by a 2004 study published in The Journal of Sports Sciences. The study analyzed testosterone and different types of strength among men who were elite amateur weight lifters and cyclists or physically fit non-athletes. Weight lifters had higher testosterone than cyclists and showed more explosive strength. But the cyclists, who had lower testosterone than both other groups, scored much higher than the others on “maximal workload,” an endurance type of strength. Across the three groups, there was no relationship between testosterone and explosive strength, and a negative relationship between testosterone and maximal workload. Though small, that study isn’t an outlier: Similar complex patterns of mixed, positive and negative relationships with testosterone are found throughout the literature, involving a wide range of sports.
In other words, the authors lard their argument with a straw man that ignores the main point — testosterone drives male puberty, which increases muscle mass, bone density and size, and a host of other side effects beneficial to athletic achievement. They also conveniently omit the fact that
Caster Semenya is genetically male, despite being phenotypically female:
1) Caster Semenya Has XY Chromosomes
It’s
absolutely mind-boggling that virtually every major outlet in the world
reporting the Court of Arbitration for Sport ruling yesterday has failed
to mention one of the most important facts of the entire case. Caster
Semenya has XY chromosomes. It was generally accepted by people
following the case closely that Semenya was XY, but now it’s been
confirmed as fact since the CAS press release specifically says,
“The DSD covered by the Regulations are limited to athletes with ’46 XY
DSD’ – i.e. conditions where the affected individual has XY
chromosomes.” If she wasn’t XY, the IAAF’s regulations wouldn’t apply to
her and she’d have no reason to challenge them.
(In case you forgot what you learned in junior high biology, typically females have XX chromosomes while males are XY).
How the Associated Press, Reuters, NY Times, NPR, Washington Post, and BBC
could all leave this CRUCIAL fact out of their reporting is beyond me.
Not a single one of them mentioned it at all. It should have been in the
lead paragraph of every story so people like my mother, who sent me a
confused email after she saw an article on Semenya, can really
understand what this is all about. Instead, the closest we get to the
truth was that some of the articles talked about how Semenya has
intersex “traits” or “characteristics.” Let’s be real, if you are an XY
woman, you are the very definition of what virtually everyone would
think of as intersex.
The woke academy has taken over science discussion.
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